Why cholecystokinin and gastrin are also incretins

Jens F. Rehfeld

doi:10.1097/XCE.0000000000000095

Why cholecystokinin and gastrin are also incretins

Jens F. Rehfeld

Institut for Klinisk Medicin

6 Citationer (Scopus)

Abstract

This review argues that cholecystokinin (CCK) and gastrin are incretins. The insulin cells are equipped with CCK 2 /gastrin receptors. CCK/gastrin peptides stimulate insulin secretion and potentiate the incretin effect of glucagon-like peptide-1. CCK/gastrin and insulin are released in significant amounts during normal mixed meals even at modest changes in blood glucose concentrations.

Originalsprog	Engelsk
Tidsskrift	Cardiovascular Endocrinology (Online)
Vol/bind	5
Udgave nummer	3
Sider (fra-til)	99-101
ISSN	2162-688X
DOI	https://doi.org/10.1097/XCE.0000000000000095
Status	Udgivet - sep. 2016

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abstract = "This review argues that cholecystokinin (CCK) and gastrin are incretins. The insulin cells are equipped with CCK 2 /gastrin receptors. CCK/gastrin peptides stimulate insulin secretion and potentiate the incretin effect of glucagon-like peptide-1. CCK/gastrin and insulin are released in significant amounts during normal mixed meals even at modest changes in blood glucose concentrations.",

author = "Rehfeld, {Jens F.}",

year = "2016",

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AB - This review argues that cholecystokinin (CCK) and gastrin are incretins. The insulin cells are equipped with CCK 2 /gastrin receptors. CCK/gastrin peptides stimulate insulin secretion and potentiate the incretin effect of glucagon-like peptide-1. CCK/gastrin and insulin are released in significant amounts during normal mixed meals even at modest changes in blood glucose concentrations.

U2 - 10.1097/XCE.0000000000000095

DO - 10.1097/XCE.0000000000000095

M3 - Review

SN - 2162-688X

VL - 5

SP - 99

EP - 101

JO - Cardiovascular Endocrinology

JF - Cardiovascular Endocrinology

IS - 3

ER -

Why cholecystokinin and gastrin are also incretins

Abstract

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