Udvikling af refraktaert vasodilatorisk shock og permanent høretab efter svaer lithiumforgiftning

Ulla Plenge; Ann M Møller

Udvikling af refraktaert vasodilatorisk shock og permanent høretab efter svaer lithiumforgiftning

Translated title of the contribution: [Development of sustained vasodilatory shock and permanent loss of hearing after severe lithium carbonate poisoning]

Ulla Plenge, Ann M Møller

2 Citations (Scopus)

Abstract

A case of severe lithium carbonate self-poisoning is described, presenting with sustained vasodilatory shock, DIC and neurologic manifestations. Lengthy and repeated haemodialysis was required to lower lithemia to non-toxic levels in accordance with which the haemodynamic instability and multi organ dysfunction ceased. The patient survived with marked loss of hearing, tinnitus and vertigo as the only sequelae. We discuss the mechanism of lithium's inhibitory effect on the phosphoinositide cascade as the possible explanation for the vasodilatory shock as well as the hearing deficiency.

Translated title of the contribution	[Development of sustained vasodilatory shock and permanent loss of hearing after severe lithium carbonate poisoning]
Original language	Danish
Journal	Ugeskrift for Laeger
Volume	170
Issue number	5
Pages (from-to)	354
ISSN	0041-5782
Publication status	Published - 28 Jan 2008

Cite this

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abstract = "A case of severe lithium carbonate self-poisoning is described, presenting with sustained vasodilatory shock, DIC and neurologic manifestations. Lengthy and repeated haemodialysis was required to lower lithemia to non-toxic levels in accordance with which the haemodynamic instability and multi organ dysfunction ceased. The patient survived with marked loss of hearing, tinnitus and vertigo as the only sequelae. We discuss the mechanism of lithium's inhibitory effect on the phosphoinositide cascade as the possible explanation for the vasodilatory shock as well as the hearing deficiency.",

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AU - Møller, Ann M

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Y1 - 2008/1/28

N2 - A case of severe lithium carbonate self-poisoning is described, presenting with sustained vasodilatory shock, DIC and neurologic manifestations. Lengthy and repeated haemodialysis was required to lower lithemia to non-toxic levels in accordance with which the haemodynamic instability and multi organ dysfunction ceased. The patient survived with marked loss of hearing, tinnitus and vertigo as the only sequelae. We discuss the mechanism of lithium's inhibitory effect on the phosphoinositide cascade as the possible explanation for the vasodilatory shock as well as the hearing deficiency.

AB - A case of severe lithium carbonate self-poisoning is described, presenting with sustained vasodilatory shock, DIC and neurologic manifestations. Lengthy and repeated haemodialysis was required to lower lithemia to non-toxic levels in accordance with which the haemodynamic instability and multi organ dysfunction ceased. The patient survived with marked loss of hearing, tinnitus and vertigo as the only sequelae. We discuss the mechanism of lithium's inhibitory effect on the phosphoinositide cascade as the possible explanation for the vasodilatory shock as well as the hearing deficiency.

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SN - 0041-5782

VL - 170

SP - 354

JO - Ugeskrift for Laeger

JF - Ugeskrift for Laeger

IS - 5

ER -

Udvikling af refraktaert vasodilatorisk shock og permanent høretab efter svaer lithiumforgiftning

Abstract

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