Paternal high-fat diet transgenerationally impacts hepatic immunometabolism

Thais de Castro Barbosa, Petter S. Alm, Anna Krook, Romain Barrès, Juleen R. Zierath*

*Corresponding author for this work
    5 Citations (Scopus)

    Abstract

    Paternal preconceptional high-fat diet (HFD) alters whole-body glucose and energy homeostasis over several generations, which may be mediated by altered transcriptomic profiles of metabolic organs. We investigated the effect of paternal HFD on the hepatic transcriptomic and metabolic signatures of female grand-offspring. Paternal HFD strongly impacted the liver transcriptome of the second-generation offspring. Gene set enrichment analysis (GSEA) revealed grandpaternal-HFD altered the TNF-α signaling via NFκB pathway, independent of the grand-offspring's diet. Reduction in the hepatic cytokine levels, including the TNF-α, as well as NFκB content and activity, suggest that the basal inflammatory response in F2 rats is disturbed. GSEA also show altered expression of various genes annotated to the fatty acid metabolism. Grandpaternal-HFD reduced G0/G1 switch gene 2 (G0S2) expression, concomitant with reduced hepatic triglyceride content in F2 rats. In conclusion, the hepatic transcriptome is altered in grand-offspring from HFD-fed grandfathers. Altered TNF-α/NFκB signaling and levels of inflammatory cytokines indicate grand-paternal HFD impacts hepatic immunometabolism. Overall, our findings indicate that paternal exposure to environmental factors transgenerationally reprograms metabolism in a tissue-specific manner, affecting the development and health of future generations.—De Castro Barbosa, T., Alm, P. S., Krook, A., Barrès, R., Zierath, J. R. Paternal high-fat diet transgenerationally impacts hepatic immunometabolism. FASEB J. 33, 6269–6280 (2019). www.fasebj.org.

    Original languageEnglish
    JournalFASEB Journal
    Volume33
    Issue number5
    Pages (from-to)6269-6280
    ISSN0892-6638
    DOIs
    Publication statusPublished - 1 May 2019

    Keywords

    • metabolism
    • epigenetics
    • cytokines
    • liver

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