Hypothesis: type I toxin - antitoxin genes enter the persistence field-a feedback mechanism explaining membrane homoeostasis

10 Citations (Scopus)
Original languageEnglish
Article number20160189
JournalRoyal Society of London. Philosophical Transactions B. Biological Sciences
Volume371
Issue number1707
Number of pages9
ISSN0962-8436
DOIs
Publication statusPublished - 1 Nov 2016

Keywords

  • bacterial persistence
  • toxin
  • antitoxin
  • HokB
  • RNase E
  • membrane homoeostasis

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