Endogenous and recombinant type I interferons and disease activity in multiple sclerosis

Finn Sellebjerg, Martin Krakauer, Signe Limborg, Dan Hesse, Henrik Lund, Annika Langkilde, Helle Bach Søndergaard, Per Soelberg Sørensen

12 Citations (Scopus)

Abstract

Although treatment of multiple sclerosis (MS) with the type I interferon (IFN) IFN-β lowers disease activity, the role of endogenous type I IFN in MS remains controversial. We studied CD4+ T cells and CD4+ T cell subsets, monocytes and dendritic cells by flow cytometry and analysed the relationship with endogenous type I IFN-like activity, the effect of IFN-β therapy, and clinical and magnetic resonance imaging (MRI) disease activity in MS patients. Endogenous type I IFN activity was associated with decreased expression of the integrin subunit CD49d (VLA-4) on CD4+CD26 high T cells (Th1 helper cells), and this effect was associated with less MRI disease activity. IFN-β therapy reduced CD49d expression on CD4+CD26 high T cells, and the percentage of CD4+CD26 high T cells that were CD49d high correlated with clinical and MRI disease activity in patients treated with IFN-β. Treatment with IFN-β also increased the percentage of CD4+ T cells expressing CD71 and HLA-DR (activated T cells), and this was associated with an increased risk of clinical disease activity. In contrast, induction of CD71 and HLA-DR was not observed in untreated MS patients with evidence of endogenous type IFN I activity. In conclusion, the effects of IFN-β treatment and endogenous type I IFN activity on VLA-4 expression are similar and associated with control of disease activity. However, immune-activating effects of treatment with IFN-β may counteract the beneficial effects of treatment and cause an insufficient response to therapy.

Original languageEnglish
JournalP L o S One
Volume7
Issue number6
Pages (from-to)e35927
ISSN1932-6203
DOIs
Publication statusPublished - 6 Jun 2012

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