CI^- and K⁺ Channels in Pancreatic Ductal Adenocarcinoma (PDAC)

Keyphrases

• Pancreatic Ductal Adenocarcinoma 100%
• ANO1 100%
• K+ Channels 100%
• PH Regulation 60%
• Ca2+ 60%
• KCa3.1 40%
• K+ Current 40%
• Cell Migration 40%
• Patch-clamp Recording 20%
• TMEM16A 20%
• Quantitative PCR 20%
• Capan-1 20%
• Ca2+ Homeostasis 20%
• Proliferation Migration 20%
• Channel Activator 20%
• Survival Benefit 20%
• Ion Channels 20%
• Western Blotting 20%
• Patch-clamp Technique 20%
• Biophysical Characteristics 20%
• Erlotinib 20%
• Cellular Migration 20%
• Tumor Biology 20%
• Tumor Progression 20%
• Functional Expression 20%
• Survival Rate 20%
• Cell Proliferation 20%
• Proliferation Apoptosis 20%
• High-throughput 20%
• All Cancer 20%
• Pharmacological Attributes 20%
• New Therapeutic Strategies 20%
• Gemcitabine 20%
• Patch Clamp 20%
• Large-conductance Ca2+-activated K+ Channel 20%
• Cellular Processes 20%
• Cancer Hallmarks 20%
• Tumor Cells 20%
• Specific Inhibitor 20%

Biochemistry, Genetics and Molecular Biology

• Potassium Channel 100%
• ANO1 100%
• Cell Migration 60%
• Cancer Cell 40%
• Potassium Current 40%
• Patch Clamp 40%
• Survival Rate 20%
• Conductance 20%
• Tumor Progression 20%
• Real-Time Polymerase Chain Reaction 20%
• Patch Clamp Technique 20%
• Programmed Cell Death 20%
• Western Blot 20%
• Gemcitabine 20%
• Homeostasis 20%
• Ion Channel 20%
• Wound Healing Assay 20%
• Gene Inactivation 20%
• Cell Proliferation 20%

Pharmacology, Toxicology and Pharmaceutical Science

• Potassium Channel 100%
• Pancreas Adenocarcinoma 100%
• Calcium Ion 100%
• Pancreas Cancer 40%
• Potassium Ion 40%
• Neoplasm 40%
• Gemcitabine 20%
• Tumor Growth 20%
• Survival Rate 20%
• Malignant Neoplasm 20%
• Western Blot 20%
• Wound Healing 20%
• Cellular Processes 20%
• Ion Channel 20%