Ca2+ induces spontaneous dephosphorylation of a novel P5A-type ATPase

Danny Mollerup Sørensen, Annette B. Møller, Mia K. Jakobsen, Michael Krogh Jensen, Peter Vangheluwe, Morten J. Buch-Pedersen, Michael Broberg Palmgren

    12 Citations (Scopus)

    Abstract

    P5 ATPases constitute the least studied group of P-type ATPases, an essential family of ion pumps in all kingdoms of life. Although P5 ATPases are present in every eukaryotic genome analyzed so far, they have remained orphan pumps, and their biochemical function is obscure. We show that a P5A ATPase from barley, HvP5A1, locates to the endoplasmic reticulum and is able to rescue knock-out mutants of P5A genes in both Arabidopsis thaliana and Saccharomyces cerevisiae. HvP5A1 spontaneously forms a phosphorylated reaction cycle intermediate at the catalytic residue Asp-488, whereas, among all plant nutrients tested, only Ca2+ triggers dephosphorylation. Remarkably, Ca2+-induced dephosphorylation occurs at high apparent [Ca 2+] (Ki = 0.25 mM) and is independent of the phosphatase motif of the pump and the putative binding site for transported ligands located in M4. Taken together, our results rule out that Ca2+ is a transported substrate but indicate the presence of a cytosolic low affinity Ca2+-binding site, which is conserved among P-type pumps and could be involved in pump regulation. Our work constitutes the first characterization of a P5 ATPase phosphoenzyme and points to Ca2+ as a modifier of its function.

    Original languageEnglish
    JournalJournal of Biological Chemistry
    Volume287
    Issue number34
    Pages (from-to)28336-28348
    Number of pages13
    ISSN0021-9258
    DOIs
    Publication statusPublished - 17 Aug 2012

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