The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes

Michael Gaster; Klaus Brusgaard; Aa. Handberg; K. Højlund; Jørgen Wojtaszewski; H. Beck-Nielsen

doi:10.1016/j.bbrc.2004.05.109

The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes

Michael Gaster, Klaus Brusgaard, Aa. Handberg, K. Højlund, Jørgen Wojtaszewski, H. Beck-Nielsen

15 Citationer (Scopus)

Originalsprog	Engelsk
Tidsskrift	Biochemical and Biophysical Research Communications
Vol/bind	319
Udgave nummer	4
Sider (fra-til)	1235-1240
ISSN	0006-291X
DOI	https://doi.org/10.1016/j.bbrc.2004.05.109
Status	Udgivet - 2004

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10.1016/j.bbrc.2004.05.109

Citationsformater

The primary defect in glycogen synthase activity is not based on increased glycogen synthase kinase-3a activity in diabetic myotubes. / Gaster, Michael; Brusgaard, Klaus; Handberg, Aa. et al.

I: Biochemical and Biophysical Research Communications, Bind 319, Nr. 4, 2004, s. 1235-1240.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › peer review

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AU - Gaster, Michael

AU - Brusgaard, Klaus

AU - Handberg, Aa.

AU - Højlund, K.

AU - Wojtaszewski, Jørgen

AU - Beck-Nielsen, H.

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