Spontaneous prenatal ductal closure: postnatal diagnosis?

Sissel Irene Nygaard; Olav Bjorn Petersen; Ester Garne; Keld Ejvind Sørensen

doi:10.1007/s00246-008-9269-1

Spontaneous prenatal ductal closure: postnatal diagnosis?

Sissel Irene Nygaard, Olav Bjorn Petersen, Ester Garne, Keld Ejvind Sørensen

9 Citationer (Scopus)

Abstract

Prenatal ductal constriction and closure usually are related to maternal intake of prostaglandin receptor inhibitors during late gestation but may occur spontaneously. This report describes three live-born infants sharing echocardiographic features of spontaneous intrauterine ductal constriction, closure, or both, although only one had a prenatal diagnosis. Interestingly, in addition to severely hypertrophied right ventricles, all three had hyperechogenic tricuspid valve papillary muscles. Two also had mild ascending aorta dilation. These cases raise mechanistic questions because of intrauterine ductal occlusion, bright papillary muscles, and potential dilated ascending aortas.

Originalsprog	Engelsk
Tidsskrift	Pediatric Cardiology
Vol/bind	30
Udgave nummer	2
Sider (fra-til)	176-80
ISSN	0172-0643
DOI	https://doi.org/10.1007/s00246-008-9269-1
Status	Udgivet - 2009
Udgivet eksternt	Ja

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10.1007/s00246-008-9269-1

Citationsformater

@article{ed6ef62a98b442f6a740eeded9701561,

title = "Spontaneous prenatal ductal closure: postnatal diagnosis?",

abstract = "Prenatal ductal constriction and closure usually are related to maternal intake of prostaglandin receptor inhibitors during late gestation but may occur spontaneously. This report describes three live-born infants sharing echocardiographic features of spontaneous intrauterine ductal constriction, closure, or both, although only one had a prenatal diagnosis. Interestingly, in addition to severely hypertrophied right ventricles, all three had hyperechogenic tricuspid valve papillary muscles. Two also had mild ascending aorta dilation. These cases raise mechanistic questions because of intrauterine ductal occlusion, bright papillary muscles, and potential dilated ascending aortas.",

keywords = "Aorta/abnormalities, Aortic Diseases/diagnostic imaging, Ductus Arteriosus, Patent/diagnostic imaging, Female, Humans, Hypertrophy, Right Ventricular/diagnostic imaging, Infant, Newborn, Male, Papillary Muscles/abnormalities, Tricuspid Valve/abnormalities, Ultrasonography, Prenatal",

author = "Nygaard, {Sissel Irene} and Petersen, {Olav Bjorn} and Ester Garne and S{\o}rensen, {Keld Ejvind}",

year = "2009",

doi = "10.1007/s00246-008-9269-1",

language = "English",

volume = "30",

pages = "176--80",

journal = "Pediatric Cardiology",

issn = "0172-0643",

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TY - JOUR

T1 - Spontaneous prenatal ductal closure

T2 - postnatal diagnosis?

AU - Nygaard, Sissel Irene

AU - Petersen, Olav Bjorn

AU - Garne, Ester

AU - Sørensen, Keld Ejvind

PY - 2009

Y1 - 2009

N2 - Prenatal ductal constriction and closure usually are related to maternal intake of prostaglandin receptor inhibitors during late gestation but may occur spontaneously. This report describes three live-born infants sharing echocardiographic features of spontaneous intrauterine ductal constriction, closure, or both, although only one had a prenatal diagnosis. Interestingly, in addition to severely hypertrophied right ventricles, all three had hyperechogenic tricuspid valve papillary muscles. Two also had mild ascending aorta dilation. These cases raise mechanistic questions because of intrauterine ductal occlusion, bright papillary muscles, and potential dilated ascending aortas.

AB - Prenatal ductal constriction and closure usually are related to maternal intake of prostaglandin receptor inhibitors during late gestation but may occur spontaneously. This report describes three live-born infants sharing echocardiographic features of spontaneous intrauterine ductal constriction, closure, or both, although only one had a prenatal diagnosis. Interestingly, in addition to severely hypertrophied right ventricles, all three had hyperechogenic tricuspid valve papillary muscles. Two also had mild ascending aorta dilation. These cases raise mechanistic questions because of intrauterine ductal occlusion, bright papillary muscles, and potential dilated ascending aortas.

KW - Aorta/abnormalities

KW - Aortic Diseases/diagnostic imaging

KW - Ductus Arteriosus, Patent/diagnostic imaging

KW - Female

KW - Humans

KW - Hypertrophy, Right Ventricular/diagnostic imaging

KW - Infant, Newborn

KW - Male

KW - Papillary Muscles/abnormalities

KW - Tricuspid Valve/abnormalities

KW - Ultrasonography, Prenatal

U2 - 10.1007/s00246-008-9269-1

DO - 10.1007/s00246-008-9269-1

M3 - Journal article

C2 - 18779991

SN - 0172-0643

VL - 30

SP - 176

EP - 180

JO - Pediatric Cardiology

JF - Pediatric Cardiology

IS - 2

ER -

Spontaneous prenatal ductal closure: postnatal diagnosis?

Abstract

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