Matching NLR immune receptors to autoimmunity in camta3 mutants using antimorphic NLR alleles

Signe Lolle, Michael Christiaan Greeff, Klaus Petersen, Milena Edna Roux, Michael Krogh Jensen, Simon Bressendorff, Eleazar José Rodriguez Gomes, Kenneth Sømark, John Mundy, Morten Petersen*

*Corresponding author af dette arbejde
34 Citationer (Scopus)

Abstract

To establish infection, pathogens deploy effectors to modify or remove host proteins. Plant immune receptors with nucleotide-binding, leucine-rich repeat domains (NLRs) detect these modifications and trigger immunity. Plant NLRs thus guard host “guardees.” A corollary is that autoimmunity may result from inappropriate NLR activation because mutations in plant guardees could trigger corresponding NLR guards. To explore these hypotheses, we expressed 108 dominant-negative (DN) Arabidopsis NLRs in various lesion mimic mutants, including camta3, which exhibits autoimmunity. CAMTA3 was previously described as a negative regulator of immunity, and we find that autoimmunity in camta3 is fully suppressed by expressing DNs of two NLRs, DSC1 and DSC2. Additionally, expression of either NLR triggers cell death that can be suppressed by CAMTA3 expression. These findings support a model in which DSC1 and DSC2 guard CAMTA3, and they suggest that other negative regulators of immunity may similarly represent guardees.

OriginalsprogEngelsk
TidsskriftCell Host & Microbe
Vol/bind21
Udgave nummer4
Sider (fra-til)518-529
Antal sider12
ISSN1931-3128
DOI
StatusUdgivet - 2017

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