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Hypothesis: type I toxin - antitoxin genes enter the persistence field-a feedback mechanism explaining membrane homoeostasis
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Functional genomics
10
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Keyphrases
Feedback Mechanism
100%
Membrane Homeostasis
100%
(p)ppGpp
55%
Environmental Stress
22%
Toxin-antitoxin Modules
22%
Antisense RNA
22%
Positive Control
11%
Membrane Depolarization
11%
Toxin Expression
11%
Membrane Potential
11%
GTPase
11%
Membrane Protein
11%
Antibiotic Combination
11%
Stasis
11%
Persister Cells
11%
Negative Control
11%
Second Messenger
11%
Nutrient Starvation
11%
Molecular Mechanism
11%
Membrane Binding
11%
Enterobacteria
11%
Overexpression
11%
Control Mechanism
11%
Alarmone
11%
Biochemistry, Genetics and Molecular Biology
Ribonuclease E
33%
Cell Membrane Depolarization
33%
Immunology and Microbiology
Cell Membrane Depolarization
33%